Tag: Rabbit Polyclonal to CAMK5

Although alcoholic liver disease is clinically well described, the molecular basis for alcohol-induced hepatotoxicity is not well understood. (a microtubule-stabilizing drug) or trichostatin A (a deacetylase inhibitor), providers that promote microtubule acetylation in the absence of alcohol. Therefore the alcohol-induced impairment of STAT nuclear translocation can become explained by improved microtubule acetylation and stability. Only…